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Here we demonstrate that acute brain injury induces vascular damage, meningeal cell death, and the generation of reactive oxygen species (ROS) that ultimately breach the glial limitans and promote spread of the injury into the parenchyma
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The breakdown of self-tolerance to thyroid antigens leads to the onset of AITDs, a process that can be triggered by various factors, including excessive exposure to thyroid antigens, exposure to environmental antigens resembling self-antigens, polyclonal immune activation, or idiotype cross-reaction of self-antigens (17)
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